The Recommended Dosage For Nimodipine

The Recommended Dosage For Nimodipine
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The Recommended Dosage For Nimodipine
Question 59
I have seen many established ischaemic stroke patients with CTdocumented capsular infarction and hemi-hypotonia despite exaggerated
reflexes. How would you explain the hypotonia? Could it be due to a
corticorubral fibre lesion?
Question 60
Thrombolytic therapy is used in patients with a cerebral infarct within
the first 3 hours, whereas stroke by definition lasts 24 hours. So how
do we define that it is infarct and not a transient ischaemic attack (TIA)
on a CT scan within the first 3 hours in order to start tissue plasminogen
activator (tPA) treatment?
Question 61
I want to ask something about cerebrovascular accident (CVA). Can you
please tell me how we can rapidly pinpoint the exact anatomical site of the
neurological deficit using physical findings in the emergency room?
Question 62
Why do you treat dissection of the carotid artery with an anticoagulant
in the acute management of stroke secondary to dissection? To me this
seems paradoxical as it would increase the severity of dissection.
Question 63
Last week, in a neurology viva, I was asked about the indications for
heparinization in patients with a stroke. I want to know when I can stop
heparin and what test I should use for assessing its therapeutic range.
Question 64
Has heparin a role in the management of acute ischaemic stroke not
accompanied by atrial fibrillation?
Question 65
1. In the treatment of a stroke, does low-molecular-weight heparin
(LMWH) have an advantage over heparin?
2. In an ischaemic stroke in evolution, for how long should heparin be
administered?
Question 66
Can streptokinase be used in acute cerebral infarction and, if so, what is
the dose?
Question 67
The Recommended Dosage For Nimodipine
There seems now to be a consensus about starting aspirin therapy in
acute ischaemic strokes as early as possible. Why has this changed from
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past recommendations to avoid aspirin early (during the first 48 hours)
during the ischaemic stroke on the pretext that it could convert an
ischaemic infarct into a haemorrhagic one, thus increasing the dangers of
complications like cerebral oedema and raised intracranial pressure? If
both opinions are based on clinical trials, what is the significance of the
much hyped ‘evidence-based medicine’?
Question 68
I understand that a loading dose of clopidogrel 600–900 mg can be given
to ischaemic stroke in evolution and can stop the evolving deficit. Would
you agree?
Question 69
Is there any rationale for giving patients with recurrent strokes a
combination of aspirin and anticoagulant?
Question 70
1. Does a dipyridamol–aspirin combination have any superiority over
aspirin alone in the secondary prevention of a stroke?
2. Is an aspirin plus anticoagulant combination superior to a dipyridamol—
aspirin combination in the treatment of recurrent ischaemic stroke not
controlled by aspirin alone?
Question 71
1. Is it safe to give piracetam to patients with primary intracerebral
haemorrhage? Does it have a neuroprotective effect?
2. Is it safe to give a patient with excessively high blood pressure
(as a sequela to recent primary intracerebral haemorrhage)
angiotensin-converting enzyme inhibitors to lower the blood
pressure?
3. Is it indicated to give piracetam or vincamine to a patient with middle
cerebral artery territorial infarction? Do these have any neuroprotective
effect?
Question 72
What is the mechanism by which subarachnoid haemorrhage is
associated with subhyaloid haemorrhages on fundus examination, and
how can cerebrospinal fluid (CSF) gain access to the subhyaloid space
inside the eye?
Question 73
The Recommended Dosage For Nimodipine
What is the recommended dosage for nimodipine given intravenously in
cases of subarachnoid haemorrhage, and when should the treatment
start? For how long should the dose be continued?
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Question 74
I read recently that hyperuricaemia has something to do with stroke? Is it
recommended to give allopurinol to stroke patients irrespective of their
serum uric acid?
Question 75
Is there a link between hyperuricaemia (although asymptomatic) and
atherosclerosis and cerebral ischaemic stroke?
Question 76
Are phenytoin and carbamazepine indicated in myoclonus, occasionally
seen in ischemic strokes?
Question 77
What is the treatment of chorea or action myoclonus resulting from
embolic stroke to the area of basal ganglia? Does the L-dopa that is given
by some neurologists improve the condition? Does valproate have a role
if the case is action myoclonus?
Question 78
How should a patient with a haemorrhagic cerebrovascular accident
be managed while also having an extensive inferior wall myocardial
infarction?
Question 79
What is the best way to manage cortical vein thrombosis? If heparin is to
be used, what is the recommended dosage and how long should this
treatment last?
Question 80
In the case of cortical vein thrombosis, for how long should
anticoagulation be continued?
Question 81
How long should antiepileptic treatment be continued for a stroke
patient who has the first seizure within the first 24 hours of the
stroke?
Question 82
For how long should antiepileptic drugs be given to patients having their
first seizure within the first week of their cerebrovascular stroke?
Question 83
What are the causes of epilepsy with a normal electroencephalogram
(EEG), other than metabolic causes? Could epilepsy due to CNS causes
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